Search results for "Estrogen Receptors"

showing 10 items of 13 documents

miRNA as New Regulatory Mechanism of Estrogen Vascular Action

2018

The beneficial effects of estrogen on the cardiovascular system have been reported extensively. In fact, the incidence of cardiovascular diseases in women is lower than in age-matched men during their fertile stage of life, a benefit that disappears after menopause. These sex-related differences point to sexual hormones, mainly estrogen, as possible cardiovascular protective factors. The regulation of vascular function by estrogen is mainly related to the maintenance of normal endothelial function and is mediated by both direct and indirect gene transcription through the activity of specific estrogen receptors. Some of these mechanisms are known, but many remain to be elucidated. In recent …

0301 basic medicineEstrogen receptorReview030204 cardiovascular system & hematologyBioinformaticsEpigenesis Geneticlcsh:Chemistry0302 clinical medicinelcsh:QH301-705.5Spectroscopyestrogen receptorsGeneral MedicineComputer Science ApplicationsMenopauseReceptors EstrogenRNA InterferenceDisease Susceptibilitymedicine.drug_classCèl·lulesBiologyepigenetic regulationCatalysisCardiovascular Physiological PhenomenaInorganic Chemistry03 medical and health sciencesestradiolmicroRNAmedicineAnimalsHumansEpigeneticsPhysical and Theoretical ChemistryMolecular BiologyGenemiRNAReceptors d'hormonesMechanism (biology)Organic ChemistryEndothelial CellsEstrogensmedicine.diseaseMicroRNAs030104 developmental biologylcsh:Biology (General)lcsh:QD1-999Gene Expression RegulationEstrogenBlood VesselsFunction (biology)Genètica
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The analysis of estrogen receptor-α positive breast cancer stem-like cells unveils a high expression of the serpin proteinase inhibitor PI-9: Possibl…

2016

Abstract Breast cancer stem cells seem to play important roles in breast tumor recurrence and endocrine therapy resistance, although the underlying mechanisms have not been well established. Moreover, in some tumor systems the immunosurveillance failure against cancer cells has been related to the presence of the granzyme B inhibitor PI-9. This study explored the status of PI-9 in tumorspheres isolated from estrogen receptor-α positive (ERα+) breast cancer MCF7 cells. Studies were performed in tertiary tumorspheres which possess high levels of stemness markers (Nanog, Oct3/4 and Sox2) and self-renewal ability. The exposure to estrogens (17-β estradiol and genistein) increased the number and…

0301 basic medicineHomeobox protein NANOGReceptors CXCR4Cancer Researchmedicine.medical_specialtyEstrogen receptorBreast NeoplasmsBiologyp38 Mitogen-Activated Protein KinasesGranzymes03 medical and health sciences0302 clinical medicineBreast cancerSOX2Internal medicineserpin proteinase inhibitor 9 breast cancer stem-like cells breast cancer estrogen receptorsSettore BIO/10 - BiochimicamedicineHumansSerpinsCell ProliferationEstrogen Receptor alphaCancermedicine.diseaseGenisteinGene Expression Regulation NeoplasticImmunosurveillance030104 developmental biologyEndocrinologyOncology030220 oncology & carcinogenesisCancer cellMCF-7 CellsNeoplastic Stem CellsCancer researchFemaleNeoplasm Recurrence LocalStem cellSignal Transduction
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MicroRNA as crucial regulators of gene expression in estradiol-treated human endothelial cells.

2018

Background/Aims: Estrogen signalling plays an important role in vascular biology as it modulates vasoactive and metabolic pathways in endothelial cells. Growing evidence has also established microRNA (miRNA) as key regulators of endothelial function. Nonetheless, the role of estrogen regulation on miRNA profile in endothelial cells is poorly understood. In this study, we aimed to determine how estrogen modulates miRNA profile in human endothelial cells and to explore the role of the different estrogen receptors (ERα, ERβ and GPER) in the regulation of miRNA expression by estrogen. Methods: We used miRNA microarrays to determine global miRNA expression in human umbilical vein endothelial cel…

0301 basic medicinePhysiologymedicine.drug_classEndothelial cellsCèl·lulesDown-RegulationEstrogen receptorEstrogen receptorsBiologylcsh:PhysiologyEpigenetic regulationReceptors G-Protein-Coupledlcsh:Biochemistry03 medical and health sciencesDownregulation and upregulationmicroRNAGene expressionHuman Umbilical Vein Endothelial CellsmedicineCluster AnalysisHumanslcsh:QD415-436EpigeneticsCells CulturedOligonucleotide Array Sequence AnalysisPrincipal Component AnalysisReceptors d'hormoneslcsh:QP1-981EstradiolGene Expression ProfilingUp-RegulationCell biologyGene expression profilingMicroRNAsMetabolic pathway030104 developmental biologyReceptors EstrogenEstrogenMiRNA
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Dietary xenoestrogens differentially impair 3T3-L1 preadipocyte differentiation and persistently affect leptin synthesis

2008

International audience; Recent observations have highlighted adipogenesis alterations under exposure to several xenoestrogens at critical stages, and pointed at their possible involvement in the pathogenesis of obesity. However, it remains unclear whether these effects are mediated by classical estrogen receptor (ER) binding and subsequent transcriptional modulation. The aim of this study was to determine the (anti-)adipogenic impact of apigenin, bisphenol A, genistein and 17β-estradiol at the onset of adipose cell maturation, and to correlate it to their estrogenic potential. In steroid-free conditions, 3T3-L1 preadipocytes were induced to differentiate in the presence of xenoestrogens for…

Endocrinology Diabetes and Metabolismmedicine.medical_treatmentClinical BiochemistryGene ExpressionAdipose tissueEstrogen receptorBiochemistryMicechemistry.chemical_compound0302 clinical medicineEndocrinologyAdipocyte[SDV.IDA]Life Sciences [q-bio]/Food engineeringAdipocytesApigeninESTROGEN RECEPTORS0303 health sciencesEstradiolReverse Transcriptase Polymerase Chain ReactionLeptinCell Differentiation[SDV.IDA] Life Sciences [q-bio]/Food engineeringGenisteinReceptors EstrogenAdipogenesis030220 oncology & carcinogenesisIntercellular Signaling Peptides and ProteinsMolecular Medicinehormones hormone substitutes and hormone antagonistsmedicine.medical_specialty[SPI.GPROC] Engineering Sciences [physics]/Chemical and Process EngineeringXENOESTROGENSEnzyme-Linked Immunosorbent AssayBiologyModels Biological03 medical and health sciencesLEPTINPhenols3T3-L1 CellsInternal medicinemedicineAnimals[SPI.GPROC]Engineering Sciences [physics]/Chemical and Process EngineeringRNA Messengerfas ReceptorBenzhydryl CompoundsMolecular Biology030304 developmental biology3T3-L1Leptin receptorCalcium-Binding ProteinsEstrogens3T3-L1Cell BiologyADIPOGENESISPPAR gammaSteroid hormoneEndocrinologychemistry
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Isomer-nonspecific action of dichlorodiphenyltrichloroethane on aryl hydrocarbon receptor and G-protein-coupled receptor 30 intracellular signaling i…

2014

Abstract Extended residual persistence of the pesticide dichlorodiphenyltrichloroethane (DDT) raises concerns about its long-term neurotoxic effects. Little is known, however, about DDT toxicity during the early stages of neural development. This study demonstrated that DDT-induced apoptosis of mouse embryonic neuronal cells is a caspase-9-, caspase-3-, and GSK-3β-dependent process, which involves p,p’ -DDT-specific impairment of classical ERs. It also provided evidence for DDT-isomer-nonspecific alterations of AhR- and GPR30-mediated intracellular signaling, including changes in the levels of the receptor and receptor-regulated mRNAs, and also changes in the protein levels of the receptors…

GPR30Time FactorsGSK-3 betaEstrogen receptorApoptosisStimulationBiochemistryReceptors G-Protein-CoupledGlycogen Synthase Kinase 3MiceEndocrinologyneurotoxicityestrogenReceptorCells CulturedNeuronsbiologyCaspase 3estrogen receptorsCaspase InhibitorsCell biologycaspasesReceptors EstrogenQuinolinesGPERNeural developmentSignal Transductionmedicine.medical_specialtyAryl hydrocarbon receptor nuclear translocatorneuronal cell culturesDDT17-beta-estradiolIsomerismbeta-NaphthoflavoneInternal medicineparasitic diseasesCytochrome P-450 CYP1A1medicineAnimalsBcl-2BenzodioxolesRNA MessengerMolecular BiologyG protein-coupled receptorBenzoflavonesGlycogen Synthase Kinase 3 betaL-Lactate DehydrogenaseAryl hydrocarbon receptorPyrimidinesEndocrinologyReceptors Aryl Hydrocarbonbiology.proteinPyrazolesMolecular and Cellular Endocrinology
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Isoflavonoid-based bone-sparing treatments exert a low activity on reproductive organs and on hepatic metabolism of estradiol in ovariectomized rats

2007

International audience; The use of soy isoflavones is a potential alternative to hormone replacement therapy in post-menopausal bone-loss prevention. Nevertheless, phytoestrogens can target other organs and may disrupt cell proliferation, or could modify endogenous steroid hormone metabolism. These mechanisms could be linked to an increased risk of developing cancer. We therefore studied the possible side effects of such treatments in an experimental model of menopause. Forty adult female Wistar rats were ovariectomized and fed with a genistein-, daidzein- or equol-supplemented diet at bone-sparing levels (10 mg/kg BW/day) for 3 months. The estrogenic effects were assessed by histological a…

GenisteinEstrogen receptorToxicologychemistry.chemical_compound0302 clinical medicineCytochrome P-450 Enzyme SystemBone Density[SDV.IDA]Life Sciences [q-bio]/Food engineeringESTROGEN RECEPTORS0303 health sciencesEstradiolfood and beveragesOrgan SizeEquolGenistein3. Good healthCYTOCHROME P450SOY ISOFLAVONEHormone receptor030220 oncology & carcinogenesisVaginaMicrosomes LiverFemaleMenopauseEQUOLmedicine.medical_specialtymedicine.drug_classOvariectomyPhytoestrogensBiology03 medical and health sciencesProliferating Cell Nuclear AntigenInternal medicinemedicineUTEROTROPHYAnimals[SPI.GPROC]Engineering Sciences [physics]/Chemical and Process EngineeringRats Wistar030304 developmental biologyPharmacologyUterusDaidzeinIsoflavonesRatsDisease Models AnimalEndocrinologyGene Expression RegulationchemistryEstrogenESTRADIOL METABOLISMOsteoporosisPhytoestrogensSteroid hormone metabolism
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Molecular mechanisms mediating the neuroprotective role of the selective estrogen receptor modulator, bazedoxifene, in acute ischemic stroke: A compa…

2017

As the knowledge on the estrogenic system in the brain grows, the possibilities to modulate it in order to afford further neuroprotection in brain damaging disorders so do it. We have previously demonstrated the ability of the selective estrogen receptor modulator, bazedoxifene (BZA), to reduce experimental ischemic brain damage. The present study has been designed to gain insight into the molecular mechanisms involved in such a neuroprotective action by investigating: 1) stroke-induced apoptotic cell death; 2) expression of estrogen receptors (ER) ERα, ERβ and the G-protein coupled estrogen receptor (GPER); and 3) modulation of MAPK/ ERK1/2 and PI3K/Akt signaling pathways. For comparison, …

Male0301 basic medicineMAPK/ERK pathwayIndolesSignaling pathwaysEndocrinology Diabetes and MetabolismClinical BiochemistryEstrogen receptorApoptosisEstrogen receptorsSecond Messenger SystemsBiochemistryBrain IschemiaReceptors G-Protein-Coupled0302 clinical medicineEndocrinologyPhosphatidylinositol PhosphatesCerebral CortexNeuronsEstradiolNeuroprotectionStrokeNeuroprotective AgentsSelective estrogen receptor modulatorReperfusion InjuryMolecular MedicineSelective estrogen receptor modulatorsGPERmedicine.medical_specialtyMAP Kinase Signaling Systemmedicine.drug_classAcute ischemic strokeNerve Tissue ProteinsBazedoxifeneBiologyNeuroprotection03 medical and health sciencesInternal medicinemedicineAnimalsEstrogen Receptor betaRats WistarMolecular BiologyProtein kinase BPI3K/AKT/mTOR pathwayEstrogen Receptor alphaEstrogensCell BiologyEstrogen030104 developmental biologyEndocrinologyEstrogen030217 neurology & neurosurgeryThe Journal of Steroid Biochemistry and Molecular Biology
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Expression of sexual hormones receptors in oral squamous cell carcinoma.

2011

Sexual hormones play an important role in expression of genes involved in a wide variety of biological and neoplastic processes. The information on Estrogen Receptors (ER) expression in non-target tissues is very few and, in particular, the studies in head and neck tumors are still controversial. Recent studies analyzed the role of Tamoxifen (TAM) on Oral Squamous Cell Carcinoma (OSCC) lines in relation to the presence/absence of ER. The purpose of the present study was to evaluate the expression of sexual hormones receptors mRNAs, in particular Estrogen Receptor alpha (ERα) and Androgen Receptor (AR) mRNA in OSCC tissues. The study group comprised 20 samples of OSCC, harvested from 20 oth…

OncologyAdultMalemedicine.medical_specialtyImmunologyEstrogen receptorBiologyOral Squamous Cell CarcinomaSettore MED/28 - Malattie OdontostomatologicheEstrogen ReceptorsInternal medicinemedicineCarcinomaImmunology and AllergyHumansAndrogen Receptors; Estrogen Receptors; Oral Squamous Cell Carcinoma;Oral mucosaReceptorAgedPharmacologyOral squamous cell carcinoma estrogen receptorsAndrogen receptorEstrogen Receptor alphaCancerMiddle Agedmedicine.diseaseAndrogen receptormedicine.anatomical_structureReceptors AndrogenCase-Control StudiesCancer researchCarcinoma Squamous CellFemaleMouth NeoplasmsEstrogen receptor alphaTamoxifenmedicine.drugSexual hormones OSCCAndrogen Receptors
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Nuclear and cytoplasmic interaction of pRb2/p130 and ER-β in MCF-7 breast cancer cells

2006

Estrogens exhibit important biological functions and influence several pathological processes of hormone-dependent diseases. The biological actions of estrogens require their interaction with two estrogen receptors (ER-alpha and ER-beta), which are ligand-dependent transcription factors. ER-alpha and ER-beta exhibit distinct tissue expression patterns as well as show different patterns of gene regulation. In addition, it has been suggested that ER-beta works as a counter partner of ER-alpha through inhibition of the transactivating functions of ER-alpha. For instance, ER-beta seems to play a different role in breast tumorigenesis than ER-alpha, as ER-beta decreased expression in breast canc…

OncologyCytoplasmmedicine.medical_specialtyMolecular Sequence DataEstrogen receptorBreast NeoplasmsEstrogen receptorsmedicine.disease_causeBreast cancerBreast cancerCancer stem cellCell Line TumorInternal medicinemedicineEstrogen Receptor betaHumansImmunoprecipitationGene silencingAmino Acid SequenceTranscription factorBreast cancer; Estrogen receptors; Estrogens; pRb2/130Cell NucleusRegulation of gene expressionRetinoblastoma-Like Protein p130business.industryEstrogensHematologymedicine.diseaseOncologyMCF-7Cancer researchbusinessCarcinogenesispRb2/130
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Possible regulatory mechanisms responsible for the high expression of serpin protease inhibitor PI-9 in ER+ -derived breast cancer stem cells.

2015

Breast cancer (BC) is the most common endocrine cancer and the second leading cause of cancer-related death in women. About 75% of BCs expresses high levels of estrogen receptors that sustain the tumor growth. Moreover, in BC estrogens prevent apoptosis induced by granzyme B released by cytotoxic T lymphocytes and NK cells through the production of the granzyme B inhibitor PI-9. As a consequence, cancer cells acquire the ability to escape immune surveillance’s signaling. Although some studies explored the role of PI-9 in BC cells, its presence has not been investigated in cancer stem cells so far. In this research, tertiary tumorspheres were obtained from estrogen receptor-alfa positive (ER…

Serpin proteinase inhibitor 9 breast cancer stem-like cells breast cancer estrogen receptors.
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